Hemo Guard Supreme


Hemo Guard Supreme™ was developed for people who may be prone to hypercoagulation. Hypercoagulation is a condition where fibrin builds up in the various blood vessels. In its extreme, fibrin deposition can lead to deep vein thrombosis, pulmonary embolisms, heart attacks, and stroke. In its less severe form it can possibly lead to phlebitis, fibromyalgia (by cutting off blood flow), I.B.S., chronic low grade infections where the microbes are “protected” by residing in the fibrin meshes, miscarriages, headaches, chronic pain, etc.

Traditional medicine sees the following as potential causes: immobilization, malignancy, genetic disorders, certain medications such as tamoxifen, pregnancy, supplemental estrogen, trauma, surgery, and nephrotic syndrome.

Recent research by David Berg and many others have also implicated the following in causing hypercoagulation: many forms of infection (viral, parasitic, bacterial, and fungal) as well as toxic chemicals and metals [1, 2]. The fibrin formation may be the body’s attempt to wall off the microbes.

It is theorized by some that these newer recognized causes may account for many cases of pulmonary embolisms that were previously labeled as idiopathic.

To cause hypercoagulation there may be an excess of prothrombin, thrombin or fibrinogen or a deficiency or fibrin breakdown chemicals: plasmin, plasminogen, tissue plasminogen activator or a deficiency of antithrombin III.

Pharmaceutical intervention can be very effective but in some people can lead to serious side effects (typically bleeding events) [3]. The drugs though can be life saving and you should follow your physician’s advice.

In less severe cases, with the consent of your physician, there are certain herbs and other lifestyle changes that can discourage hypercoagulation.

Hemo Guard is a combination of some of the more effective agents. It contains the following:

1) Ginkgo biloba: Gingko has been used for many years for memory enhancement, increased cognition, and an increased ability to pay attention [4]. One property reported in a study in China is that it is a potent antagonist of platelet activating factor and thus may help prevent blood clots and hypercoagulation [5]. The World Health Organization also reported that is helpful in treating cerebrovascular insufficiency as well as peripheral artery occlusive disease [6]. Recent studies also show it acting as a scavenger of free radicals and having neuroprotective affects from hypoxia [7]. It should not be used if you are on anticoagulant therapy.

2) Ginger root: Studies at the University of Maryland suggest that Ginger has blood-thinning properties by acting as an anti-thrombotic, in addition to lowering cholesterol and being anti-inflammatory [8]. A research study conducted in Australia looked at the ability of ginger to stop or reduce platelet activation and aggregation induced by arachadonic acid in human blood [9]. They also compared and contrasted the anticoagulant properties of ginger with those of aspirin [10]. The report concluded that ginger could prevent blood platelets from adhering together. This might be due to the inhibition of the enzyme COX-1 [9]. The report also concluded that ginger compounds and the derivatives of ginger are more effective as antiplatelet agents than aspirin.

In one fascinating study on dietary effects on fibrinolysis, fibrinolytic activity decreased by 18.8 per cent after administration of fatty meal. Addition of ginger along with the fat not only prevented the fall in fibrinolytic activity but actually increased it by 6.7 per cent. As compared with the fatty meal, ginger actually increased fibrinolysis by 31.5 per cent.

Ginger has many other good properties in addition to anti-coagulation. Studies have shown consuming ginger can be a way to lower blood pressure [12]. It also contains antibacterial properties that can be used to treat microbial infections such as colliform bacillus, staphylococcus epidermidis and streptococcus viridians [13]. Ginger has also shown to help with fat digestion in addition to increasing thermogenesis and decreasing satiety (hunger), working to help with weight loss [14, 15]. This is one reason why many cultures include ginger as a spice whenever they eat fatty meals [16].

3) Green Papaya: Green papaya contains three different proteolytic enzymes: Chymopapain, Papain, and Papaya Proteinase III [17]. The strong proteolytic enzymes in papaya may assist in fibrinolysis and dissolution of biofilms [18, 19, 20].

Hemo Guard should not be used if the patient is on ‘blood thinners” such as warfarin and other similar pharmaceuticals. If the patient is not on these, it may prove to be a useful tool. It may also be used to possibly prevent conditions in people who have family histories of some of the previously mentioned hypercogulation caused problems.

In many cases we see Hemo Guard helping treat microbial infections when one is on an antimicrobial regime by allowing the antimicrobials to reach the most distal areas of the body.

Other contraindications include pregnancy and latex allergies.

As with any herbal regimen, we encourage you to consult your physician to see if there is a more serious condition warranting more aggressive intervention.

DanShen Supreme also is helpful in hypercoagulation problems and should be considered as an alternative if the patient has latex sensitivities or in cases where more cardiac issues are apparent. We have used both of them successfully but would not give both simultaneously as the effect may be too strong.

Recommended Dose: 1 cap three times daily between meals. Dosage may be increased during times of acute infection to discourage hypercoagulation.


1. Berg, D., L. H. Berg, J. Couvaras, and H. Harrison. “Chronic Fatigue Syndrome And/or Fibromyalgia as a Variation of Antiphospholipid Antibody Syndrome.” Blood Coagulation & Fibrinolysis 10.7 (1999): 435-38. Print.

2. Berg D, Berg LH, Couvaras J, Harrison H. Chronic fatigue syndrome &/or fibromyalgia as a variation of antiphospholipid antibody syndrome (APS): An explanatory model and approach to laboratory diagnosis. Blood Coagulation and Fibrinolysis 1999: 10 435-438.

3. Wysowski, Diane K., Parivash Nourjah, and Lynette Swartz. “Bleeding Complications With Warfarin Use.” Archives of Internal Medicine Arch Intern Med 167.13 (2007): 1414. Web.

4. Kennedy, David O., Andrew B. Scholey, and Keith A. Wesnes. “The Dose-dependent Cognitive Effects of Acute Administration of Ginkgo Biloba to Healthy Young Volunteers.” Psychopharmacology 151.4 (2000): 416-23. Web.

5. Lamant, Valerie, Gerard Mauco, Pierre Braquet, Hugues Chap, and Louis Douste-Blazy. “Inhibition of the Metabolism of Platelet Activating Factor (PAF-acether) by Three Specific Antagonists from Ginkgo Biloba.” Biochemical Pharmacology 36.17 (1987): 2749-752. Web.

6. Horsch, S., and C. Walther. “Ginkgo Biloba Special Extract EGb 761 in the Treatment of Peripheral Arterial Occlusive Disease (PAOD) – a Review Based on Randomized, Controlled Studies.” Int. Journal of Clinical Pharmacology and Therapeutics CP 42.02 (2004): 63-72. Web.

7. Smith, Paul F., Karyn Maclennan, and Cynthia L. Darlington. “The Neuroprotective Properties of the Ginkgo Biloba Leaf: A Review of the Possible Relationship to Platelet-activating Factor (PAF).” Journal of Ethnopharmacology 50.3 (1996): 131-39. Web.

8. Thomson, M., K.k. Al-Qattan, S.m. Al-Sawan, M.a. Alnaqeeb, I. Khan, and M. Ali. “The Use of Ginger (Zingiber Officinale Rosc.) as a Potential Anti-inflammatory and Antithrombotic Agent.” Prostaglandins, Leukotrienes and Essential Fatty Acids 67.6 (2002): 475-78. Web

9. Nurtjahja-Tjendraputra, Effie, Alaina J. Ammit, Basil D. Roufogalis, Van H. Tran, and Colin C. Duke. “Effective Anti-platelet and COX-1 Enzyme Inhibitors from Pungent Constituents of Ginger.” Thrombosis Research 111.4-5 (2003): 259-65. Web.

10. Stanger, Michael J., Lauren A. Thompson, Andrew J. Young, and Harris R. Lieberman. “Anticoagulant Activity of Select Dietary Supplements.”Nutrition Reviews 70.2 (2012): 107-17. Web.

11. Verma, SK, and A. Bordia. “Ginger, Fat and Fibrinolysis.” Indian Journal of Medical Sciences 55.2 (2001): 83-86. Web.

12. Ghayur, Muhammad Nabeel, and Anwarul Hassan Gilani. “Ginger Lowers Blood Pressure Through Blockade of Voltage-Dependent Calcium Channels.” Journal of Cardiovascular Pharmacology 45.1 (2005): 74-80. Web.

13. Malu, S.p, G.o Obochi, E.n Tawo, and B.e Nyong. “Antibacterial Activity and Medicinal Properties of Ginger (Zingiber Officinale).” Global Journal of Pure and Applied Sciences Glo Jnl Pure Appl Sci 15.3-4 (2009): n. pag. Web.

14. Beattie, John H., Fergus Nicol, Margaret-Jane Gordon, Martin D. Reid, Louise Cantlay, Graham W. Horgan, In-Sook Kwun, Ji-Yun Ahn, and Tae-Youl Ha. “Ginger Phytochemicals Mitigate the Obesogenic Effects of a High-fat Diet in Mice: A Proteomic and Biomarker Network Analysis.” Molecular Nutrition & Food Research Mol. Nutr. Food Res.55.S2 (2011): n. pag. Web.

15. Mansour, Muhammad S., Yu-Ming Ni, Amy L. Roberts, Michael Kelleman, Arindam Roychoudhury, and Marie-Pierre St-Onge. “Ginger Consumption Enhances the Thermic Effect of Food and Promotes Feelings of Satiety without Affecting Metabolic and Hormonal Parameters in Overweight Men: A Pilot Study.” Metabolism 61.10 (2012): 1347-352. Web.

16. Prakash, Usha Ns, and Krishnapura Srinivasan. “Fat Digestion and Absorption in Spice-pretreated Rats.” Journal of the Science of Food and Agriculture J. Sci. Food Agric. 92.3 (2011): 503-10. Web.

17. Zucker, Stanley, David J. Buttle, Martin J.h. Nicklin, and Alan J. Barrett. “The Proteolytic Activities of Chymopapain, Papain, and Papaya Proteinase III.” Biochimica Et Biophysica Acta (BBA) - Protein Structure and Molecular Enzymology 828.2 (1985): 196-204. Web.

18. Hewitt, Hermi, Yvonne Wint, Laurel Talabere, Sheryll Lopez, Ellen Bailey, Omkar Parshad, and Steve Weaver. “The Use of Papaya on Pressure Ulcers.” AJN, American Journal of Nursing 102.12 (2002): 73-77. Web.

19. Thomás, Galindo-Estrella, Hernández-Gutiérrez Rodolfo, Mateos-Díaz Juan, Sandoval-Fabián Georgina, Chel-Guerrero Luis, Rodríguez-Buenfil Ingrid, and Gallegos-Tintoré Santiago. “Proteolytic Activity in Enzymatic Extracts from Carica Papaya L. Cv. Maradol Harvest By-products.” Process Biochemistry 44.1 (2009): 77-82. Web.

20. Shivananda B.N., Lexley P.P. and Dale M., Wound healing activity of Carica Papaya L. in experimentally induced diabetic rats, Indian J. Exp. Bio., 2007, 45, 743.